Modulation of KCNQ1 alternative splicing regulates cardiac IKs and action potential repolarization

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Modulation of KCNQ1 alternative splicing regulates cardiac IKs and action potential repolarization.

BACKGROUND Slow delayed-rectifier potassium current (IKs) channels, made of the pore-forming KCNQ1 and auxiliary KCNE1 subunits, play a key role in determining action potential duration (APD) in cardiac myocytes. The consequences of drug-induced KCNQ1 splice alteration remain unknown. OBJECTIVE To study the modulation of KCNQ1 alternative splicing by amiloride and the consequent changes in IK...

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KCNQ1 autoantibodies: another way to regulate IKs.

A subset of patients with dilated cardiomyopathy (DCM) have been shown to carry increased levels of autoantibodies againstb-adrenoceptors, muscarinic receptors, Na+–K+-ATPase, or troponin I. – 4 There is increasing evidence that these autoantibodies can have important electrophysiological consequences and may contribute to the arrhythmogenic risk of DCM patients. In this edition of Cardiovascul...

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Subunit interaction determines IKs participation in cardiac repolarization and repolarization reserve.

BACKGROUND The role of IKs, the slow delayed rectifier K+ current, in cardiac ventricular repolarization has been a subject of debate. METHODS AND RESULTS We develop a detailed Markov model of IKs and its alpha-subunit KCNQ1 and examine their kinetic properties during the cardiac ventricular action potential at different rates. We observe that interaction between KCNQ1 and KCNE1 (the beta-sub...

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ژورنال

عنوان ژورنال: Heart Rhythm

سال: 2013

ISSN: 1547-5271

DOI: 10.1016/j.hrthm.2013.04.014